How would factor #1 be affected by inflammation and edema?
Please read carefully, then select the correct dropdown box to identify the factor being described.
airway resistance is defined as anything that slows the flow of air through the respiratory tract. The of the airway is the primary factor affecting it. It is the physical factor influencing pulmonary ventilation that would be the most affected by inflammation and edema. Both inflammation and edema would increase this factor by , increasing the energy and effort needed to move air into and out of the lungs.
How would factor #2 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
is a force created by the attraction between water molecules that tends to collapse the alveoli. It could be by the increase in fluid in the alveoli from edema.
How would factor #3 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
lung tissue elasticity refers to the ability of the lungs and the chest wall to stretch. If edema caused an increase in alevolar surface tension , [3] would be reduced, as these parameters share an inverse relationship similar to the relationship between volume and pressure stated by Boyle’s law.
Define both pulmonary gas exchange and tissue gas exchange.
Your pre-class from last week should help you through this (you can pull out the diagram you labeled tracking the journey of O2 an CO2)--this is a verbal version of the same exercise.
Pulmonary gas exchange is defined as the diffusion of gases between the and the . Under normal conditions, moves from the alveoli through the respiratory membrane into the capillary endothelium and then into the blood plasma, while moves in the opposite direction from the blood plasma into the capillary endothelial cell through the respiratory membrane and finally into the alveolar sac.
Tissue gas exchange is defined as the exchange of O2 and CO2. Under normal conditions, moves from the blood into the cells of the tissues, and , a waste product of cellular metabolism, moves from the cells of the tissue into the blood plasma. Diffusion is a(n) process that requires that a(n) exists between locations. Under normal conditions, O2 and CO2 would move from areas of partial pressure to areas of partial pressure.
During respiratory distress, airways can become inflammed. (consider what we discussed last week about bronchitis and asthma).
what would you expect to find if you measured the partial pressure of oxygen (PO2) and carbon dioxide (PCO2) in the alveoli, the blood plasma, and the cells of the tissues?
In respiratory distress, both O2 and CO2 levels in the air in the alveoli depend on pulmonary ventilation and the integrity of larger airways upstream from the alveolus. If ventilation is minimal due to the inflammation of the airways, it leads to O2 and CO2 levels in the alveoli. gas exchange would continue between blood plasma and the cells of the tissue. Cells would continue to need to obtain for cellular respiration and to eliminate , a byproduct of metabolism. The level would rise in both blood and tissues.
What are the three factors we discussed that impact pulmonary ventilation?
How would factor #1 be affected by inflammation and edema?
Please read carefully, then select the correct dropdown box to identify the factor being described.
is defined as anything that slows the flow of air through the respiratory tract. The of the airway is the primary factor affecting it. It is the physical factor influencing pulmonary ventilation that would be the most affected by inflammation and edema. Both inflammation and edema would increase this factor by , increasing the energy and effort needed to move air into and out of the lungs.
How would factor #2 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
alveolar surface tension is a force created by the attraction between water molecules that tends to collapse the alveoli. It could be increased by the increase in fluid in the alveoli from edema.
How would factor #3 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
lung tissue elasticity refers to the ability of the lungs and the chest wall to stretch. If edema caused an increase in alevolar surface tension , [3] would be reduced, as these parameters share an inverse relationship similar to the relationship between volume and pressure stated by Boyle’s law.
Define both pulmonary gas exchange and tissue gas exchange.
Your pre-class from last week should help you through this (you can pull out the diagram you labeled tracking the journey of O2 an CO2)--this is a verbal version of the same exercise.
Pulmonary gas exchange is defined as the diffusion of gases between the and the . Under normal conditions, moves from the alveoli through the respiratory membrane into the capillary endothelium and then into the blood plasma, while moves in the opposite direction from the blood plasma into the capillary endothelial cell through the respiratory membrane and finally into the alveolar sac.
Tissue gas exchange is defined as the exchange of O2 and CO2. Under normal conditions, moves from the blood into the cells of the tissues, and , a waste product of cellular metabolism, moves from the cells of the tissue into the blood plasma. Diffusion is a(n) process that requires that a(n) exists between locations. Under normal conditions, O2 and CO2 would move from areas of partial pressure to areas of partial pressure.
During respiratory distress, airways can become inflammed. (consider what we discussed last week about bronchitis and asthma).
what would you expect to find if you measured the partial pressure of oxygen (PO2) and carbon dioxide (PCO2) in the alveoli, the blood plasma, and the cells of the tissues?
In respiratory distress, both O2 and CO2 levels in the air in the alveoli depend on pulmonary ventilation and the integrity of larger airways upstream from the alveolus. If ventilation is minimal due to the inflammation of the airways, it leads to low O2 and CO2 levels in the alveoli. gas exchange would continue between blood plasma and the cells of the tissue. Cells would continue to need to obtain for cellular respiration and to eliminate , a byproduct of metabolism. The level would rise in both blood and tissues.
How would factor #1 be affected by inflammation and edema?
Please read carefully, then select the correct dropdown box to identify the factor being described.
is defined as anything that slows the flow of air through the respiratory tract. The diameter of the airway is the primary factor affecting it. It is the physical factor influencing pulmonary ventilation that would be the most affected by inflammation and edema. Both inflammation and edema would increase this factor by , increasing the energy and effort needed to move air into and out of the lungs.
How would factor #2 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
is a force created by the attraction between water molecules that tends to collapse the alveoli. It could be by the increase in fluid in the alveoli from edema.
How would factor #3 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
lung tissue elasticity refers to the ability of the lungs and the chest wall to stretch. If edema caused an increase in alevolar surface tension , [3] would be reduced, as these parameters share an inverse relationship similar to the relationship between volume and pressure stated by Boyle’s law.
Define both pulmonary gas exchange and tissue gas exchange.
Your pre-class from last week should help you through this (you can pull out the diagram you labeled tracking the journey of O2 an CO2)--this is a verbal version of the same exercise.
Pulmonary gas exchange is defined as the diffusion of gases between the and the . Under normal conditions, moves from the alveoli through the respiratory membrane into the capillary endothelium and then into the blood plasma, while moves in the opposite direction from the blood plasma into the capillary endothelial cell through the respiratory membrane and finally into the alveolar sac.
Tissue gas exchange is defined as the exchange of O2 and CO2. Under normal conditions, moves from the blood into the cells of the tissues, and , a waste product of cellular metabolism, moves from the cells of the tissue into the blood plasma. Diffusion is a(n) process that requires that a(n) exists between locations. Under normal conditions, O2 and CO2 would move from areas of partial pressure to areas of partial pressure.
During respiratory distress, airways can become inflammed. (consider what we discussed last week about bronchitis and asthma).
what would you expect to find if you measured the partial pressure of oxygen (PO2) and carbon dioxide (PCO2) in the alveoli, the blood plasma, and the cells of the tissues?
In respiratory distress, both O2 and CO2 levels in the air in the alveoli depend on pulmonary ventilation and the integrity of larger airways upstream from the alveolus. If ventilation is minimal due to the inflammation of the airways, it leads to O2 and CO2 levels in the alveoli. gas exchange would continue between blood plasma and the cells of the tissue. Cells would continue to need to obtain for cellular respiration and to eliminate , a byproduct of metabolism. The level would rise in both blood and tissues.
How would factor #1 be affected by inflammation and edema?
Please read carefully, then select the correct dropdown box to identify the factor being described.
is defined as anything that slows the flow of air through the respiratory tract. The of the airway is the primary factor affecting it. It is the physical factor influencing pulmonary ventilation that would be the most affected by inflammation and edema. Both inflammation and edema would increase this factor by , increasing the energy and effort needed to move air into and out of the lungs.
How would factor #2 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
is a force created by the attraction between water molecules that tends to collapse the alveoli. It could be by the increase in fluid in the alveoli from edema.
How would factor #3 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
refers to the ability of the lungs and the chest wall to stretch. If edema caused an increase in , [3] would be reduced, as these parameters share an inverse relationship similar to the relationship between volume and pressure stated by Boyle’s law.
Define both pulmonary gas exchange and tissue gas exchange.
Your pre-class from last week should help you through this (you can pull out the diagram you labeled tracking the journey of O2 an CO2)--this is a verbal version of the same exercise.
Pulmonary gas exchange is defined as the diffusion of gases between the tissues and the . Under normal conditions, moves from the alveoli through the respiratory membrane into the capillary endothelium and then into the blood plasma, while moves in the opposite direction from the blood plasma into the capillary endothelial cell through the respiratory membrane and finally into the alveolar sac.
Tissue gas exchange is defined as the exchange of O2 and CO2. Under normal conditions, moves from the blood into the cells of the tissues, and , a waste product of cellular metabolism, moves from the cells of the tissue into the blood plasma. Diffusion is a(n) process that requires that a(n) exists between locations. Under normal conditions, O2 and CO2 would move from areas of partial pressure to areas of partial pressure.
During respiratory distress, airways can become inflammed. (consider what we discussed last week about bronchitis and asthma).
what would you expect to find if you measured the partial pressure of oxygen (PO2) and carbon dioxide (PCO2) in the alveoli, the blood plasma, and the cells of the tissues?
In respiratory distress, both O2 and CO2 levels in the air in the alveoli depend on pulmonary ventilation and the integrity of larger airways upstream from the alveolus. If ventilation is minimal due to the inflammation of the airways, it leads to O2 and high CO2 levels in the alveoli. gas exchange would continue between blood plasma and the cells of the tissue. Cells would continue to need to obtain for cellular respiration and to eliminate , a byproduct of metabolism. The CO2 level would rise in both blood and tissues.
What are the three factors we discussed that impact pulmonary ventilation?
How would factor #1 be affected by inflammation and edema?
Please read carefully, then select the correct dropdown box to identify the factor being described.
is defined as anything that slows the flow of air through the respiratory tract. The of the airway is the primary factor affecting it. It is the physical factor influencing pulmonary ventilation that would be the most affected by inflammation and edema. Both inflammation and edema would increase this factor by decreasing diameter , increasing the energy and effort needed to move air into and out of the lungs.
How would factor #2 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
alveolar surface tension is a force created by the attraction between water molecules that tends to collapse the alveoli. It could be decreased by the increase in fluid in the alveoli from edema.
How would factor #3 be affected by inflammation and edema?
Please read carefully, then select the correct drop down option to identify the factor being described.
refers to the ability of the lungs and the chest wall to stretch. If edema caused an increase in , [3] would be reduced, as these parameters share an inverse relationship similar to the relationship between volume and pressure stated by Boyle’s law.
Define both pulmonary gas exchange and tissue gas exchange.
Your pre-class from last week should help you through this (you can pull out the diagram you labeled tracking the journey of O2 an CO2)--this is a verbal version of the same exercise.
Pulmonary gas exchange is defined as the diffusion of gases between the tissues and the . Under normal conditions, moves from the alveoli through the respiratory membrane into the capillary endothelium and then into the blood plasma, while moves in the opposite direction from the blood plasma into the capillary endothelial cell through the respiratory membrane and finally into the alveolar sac.
Tissue gas exchange is defined as the exchange of O2 and CO2. Under normal conditions, moves from the blood into the cells of the tissues, and , a waste product of cellular metabolism, moves from the cells of the tissue into the blood plasma. Diffusion is a(n) process that requires that a(n) exists between locations. Under normal conditions, O2 and CO2 would move from areas of partial pressure to areas of partial pressure.
During respiratory distress, airways can become inflammed. (consider what we discussed last week about bronchitis and asthma).
what would you expect to find if you measured the partial pressure of oxygen (PO2) and carbon dioxide (PCO2) in the alveoli, the blood plasma, and the cells of the tissues?
In respiratory distress, both O2 and CO2 levels in the air in the alveoli depend on pulmonary ventilation and the integrity of larger airways upstream from the alveolus. If ventilation is minimal due to the inflammation of the airways, it leads to O2 and CO2 levels in the alveoli. gas exchange would continue between blood plasma and the cells of the tissue. Cells would continue to need to obtain for cellular respiration and to eliminate CO2 , a byproduct of metabolism. The level would rise in both blood and tissues.
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